Polyamines as gating molecules of inward-rectifier K+ channels

Eur J Biochem. 2000 Oct;267(19):5824-9. doi: 10.1046/j.1432-1327.2000.01669.x.

Abstract

Inward-rectifier potassium (Kir) channels comprise a superfamily of potassium (K+) channels with unique structural and functional properties. Expressed in virtually all types of cells they are responsible for setting the resting membrane potential, controlling the excitation threshold and secreting K+ ions. All Kir channels present an inwardly rectifying current-voltage relation, meaning that at any given driving force the inward flow of K+ ions exceeds the outward flow for the opposite driving force. This inward-rectification is due to a voltage-dependent block of the channel pore by intracellular polyamines and magnesium. The present molecular-biophysical understanding of inward-rectification and its physiological consequences is the topic of this review. In addition to polyamines, Kir channels are gated by intracellular protons, G-proteins, ATP and phospholipids depending on the respective Kir subfamily as detailed in the following review articles.

Publication types

  • Review

MeSH terms

  • Animals
  • Cloning, Molecular
  • Humans
  • Ion Channel Gating / drug effects*
  • Ion Transport / drug effects
  • Membrane Potentials / drug effects
  • Membrane Potentials / physiology
  • Models, Molecular
  • Polyamines / pharmacology*
  • Potassium / metabolism*
  • Potassium Channels / classification
  • Potassium Channels / drug effects*
  • Potassium Channels / physiology
  • Potassium Channels, Inwardly Rectifying*
  • Protein Conformation
  • Recombinant Fusion Proteins / drug effects
  • Recombinant Fusion Proteins / metabolism

Substances

  • Polyamines
  • Potassium Channels
  • Potassium Channels, Inwardly Rectifying
  • Recombinant Fusion Proteins
  • Potassium