Overstimulation of the alpha1B-adrenergic receptor causes a "seizure plus" syndrome

Nat Med. 2001 Feb;7(2):132. doi: 10.1038/84529.
No abstract available

Publication types

  • Comment
  • Letter

MeSH terms

  • Adrenergic alpha-Antagonists / pharmacology
  • Animals
  • Antiparkinson Agents / pharmacology
  • Brain / metabolism
  • Brain / pathology*
  • Cerebral Cortex / metabolism
  • Cerebral Cortex / pathology
  • Disease Models, Animal
  • Dopamine Agents / pharmacology
  • Epilepsy, Tonic-Clonic / metabolism
  • Epilepsy, Tonic-Clonic / pathology
  • Epilepsy, Tonic-Clonic / physiopathology
  • Humans
  • Inclusion Bodies / metabolism
  • Inclusion Bodies / pathology
  • Levodopa / pharmacology
  • Mice
  • Nerve Tissue Proteins / metabolism
  • Neurodegenerative Diseases / metabolism
  • Neurodegenerative Diseases / pathology
  • Neurodegenerative Diseases / physiopathology
  • Parkinsonian Disorders / metabolism
  • Parkinsonian Disorders / pathology
  • Parkinsonian Disorders / physiopathology
  • Prazosin / analogs & derivatives*
  • Prazosin / pharmacology
  • Receptors, Adrenergic, alpha-1 / genetics
  • Receptors, Adrenergic, alpha-1 / metabolism
  • Receptors, Adrenergic, alpha-1 / physiology*
  • Shy-Drager Syndrome / metabolism
  • Shy-Drager Syndrome / pathology*
  • Shy-Drager Syndrome / physiopathology
  • Syndrome
  • Synucleins
  • Tyrosine 3-Monooxygenase / metabolism

Substances

  • ADRA1B protein, human
  • Adra1b protein, mouse
  • Adrenergic alpha-Antagonists
  • Antiparkinson Agents
  • Dopamine Agents
  • Nerve Tissue Proteins
  • Receptors, Adrenergic, alpha-1
  • Synucleins
  • Levodopa
  • Terazosin
  • Tyrosine 3-Monooxygenase
  • Prazosin