Control of smooth muscle cell function by membrane-type matrix metalloproteinases

Ann N Y Acad Sci. 2001 Dec:947:337-40. doi: 10.1111/j.1749-6632.2001.tb03958.x.

Abstract

Vascular smooth muscle cells (SMCs) express membrane-type matrix metalloproteinases-1 and -3 (MT1- and MT3-MMPs). Expression is induced by PDGF in culture or by balloon injury in rat carotid arteries. In this study, we tried to define their functions in SMCs by transducing MT1- and MT3-MMP cDNAs into baboon-cultured SMCs, using adenoviral vectors. Overexpression of MT1-MMP increased the conversion of proMMP-2 to the activated form. In contrast, in MT3-MMP overexpressing cells, MMP-2 activation was partial. However, both MT1- and MT3-MMP overexpression elicited morphological alterations (cell rounding), which was prevented by BB94 addition. The cells, which underwent this change, showed reduced adhesion to matrices and increased migration in a Boyden chamber.

MeSH terms

  • Animals
  • Carotid Arteries / physiopathology*
  • Cell Membrane / enzymology
  • Cells, Cultured
  • Enzyme Activation
  • Genetic Vectors
  • Kinetics
  • Matrix Metalloproteinase 2 / genetics
  • Matrix Metalloproteinase 2 / metabolism
  • Matrix Metalloproteinases / genetics
  • Matrix Metalloproteinases / metabolism*
  • Matrix Metalloproteinases, Membrane-Associated
  • Metalloendopeptidases / genetics
  • Metalloendopeptidases / metabolism
  • Muscle, Smooth, Vascular / physiology*
  • Papio
  • Platelet-Derived Growth Factor / pharmacology
  • Rats
  • Transduction, Genetic

Substances

  • Platelet-Derived Growth Factor
  • Matrix Metalloproteinases
  • Matrix Metalloproteinases, Membrane-Associated
  • Metalloendopeptidases
  • Matrix Metalloproteinase 2