Protective molecular mimicry in experimental myasthenia gravis

J Neuroimmunol. 2002 May;126(1-2):99-106. doi: 10.1016/s0165-5728(02)00069-3.

Abstract

Protein databases were searched for microbial sequences that bear amino acid similarities with identified T- or B-cell epitopes within the human alpha-subunit of acetylcholine receptor (AChR). One peptide, derived from Haemophilus influenzae, exhibits 50% homology to an identified T-cell epitope of AChR alpha-subunit. This peptide was shown to have a protective effect in experimental autoimmune myasthenia gravis (EAMG). Pretreatment of rats with the mimicry peptide attenuated the induction and progression of EAMG. These effects were accompanied by a reduced T-cell response to AChR, diminished IL-2, IL-12, IFN-gamma and IL-4 levels, as well as decreased humoral response to self-AChR.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Epitopes, B-Lymphocyte / chemistry
  • Epitopes, B-Lymphocyte / immunology
  • Epitopes, T-Lymphocyte / chemistry
  • Epitopes, T-Lymphocyte / immunology
  • Female
  • Humans
  • Immune Tolerance / drug effects
  • Immune Tolerance / immunology
  • Molecular Mimicry / immunology*
  • Molecular Sequence Data
  • Myasthenia Gravis, Autoimmune, Experimental / drug therapy*
  • Myasthenia Gravis, Autoimmune, Experimental / immunology*
  • Peptide Fragments / chemistry
  • Peptide Fragments / immunology
  • Peptide Fragments / pharmacology
  • Rats
  • Rats, Inbred Lew
  • Receptors, Nicotinic / chemistry
  • Receptors, Nicotinic / immunology*
  • Th1 Cells / immunology
  • Th2 Cells / immunology

Substances

  • Epitopes, B-Lymphocyte
  • Epitopes, T-Lymphocyte
  • Peptide Fragments
  • Receptors, Nicotinic