Abstract
We investigated c-Met expression in cultured astrocytes and their regulation by cytokines. Immunocytochemistry revealed that c-Met was expressed in cultured astrocytes. Western blotting revealed that acidic and basic fibroblast growth factor (FGF) enhanced and hepatocyte growth factor (HGF) reduced c-Met expression. Reverse transcription-polymerase chain reaction revealed that FGFs and HGF enhanced c-met expression. These findings suggest that c-Met expressed in astrocytes may have important roles during the nervous system regeneration.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Animals, Newborn
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Astrocytes / physiology*
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Cells, Cultured
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Cytokines / pharmacology*
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Epidermal Growth Factor / pharmacology
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Fibroblast Growth Factor 1 / pharmacology
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Fibroblast Growth Factor 2 / pharmacology
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Hepatocyte Growth Factor / pharmacology
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Interleukin-1 / pharmacology
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Nerve Regeneration
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Proto-Oncogene Proteins c-met / genetics*
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Rats
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Rats, Wistar
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Reverse Transcriptase Polymerase Chain Reaction
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Tumor Necrosis Factor-alpha / pharmacology
Substances
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Cytokines
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Interleukin-1
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Tumor Necrosis Factor-alpha
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Fibroblast Growth Factor 2
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Fibroblast Growth Factor 1
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Epidermal Growth Factor
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Hepatocyte Growth Factor
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Proto-Oncogene Proteins c-met