Effective interferon therapy for chronic hepatitis C patients with low viral loads

Hepatogastroenterology. 2003 May-Jun;50(51):817-20.

Abstract

Background/aims: Possible short-term interferon therapy was investigated in chronic hepatitis C patients with genotype 2a or 2b and low viral-loads. Furthermore, initial changes of hepatic C virus RNA levels in early phase interferon therapy, and the number of pretreatment mutated clones at hypervariable region-1 were determined in order to upgrade interferon therapy efficacy prediction rates.

Methodology: Study subjects were 31 patients with histologically proven chronic hepatitis C, having less than 1 Meq/mL of hepatic C virus RNA levels. Daily dose was defined as 9 MU of interferon; patients with genotype lb were treated for 26 weeks, while those with genotype 2a or 2b were treated for 16 weeks.

Results: Sustained response rates showed no difference in efficacy between the 2 groups (66.7% vs. 62.5%). Response rates based on the number of hypervariable region-1 clones indicated that the fewer the number of mutated clones, more significant was the increase in efficacy. Efficacy as hepatic C virus RNA in early phase treatment showed no difference in response rates between negative and positive groups at any time point from day 1.

Conclusions: In a low viral-load group, the number of hypervariable region-1 clones was a critical factor influencing interferon therapy efficacy. Thus, 16-week interferon therapy was effective and economical.

MeSH terms

  • Adult
  • Aged
  • Antiviral Agents / administration & dosage*
  • Biopsy
  • Dose-Response Relationship, Drug
  • Drug Administration Schedule
  • Female
  • Genotype
  • Hepacivirus / drug effects
  • Hepacivirus / genetics
  • Hepatitis C, Chronic / drug therapy*
  • Hepatitis C, Chronic / pathology
  • Hepatitis C, Chronic / virology
  • Humans
  • Interferon alpha-2
  • Interferon-alpha / administration & dosage*
  • Interferon-alpha / adverse effects
  • Liver / pathology
  • Liver Function Tests
  • Male
  • Middle Aged
  • Mutation / genetics
  • RNA, Viral / blood
  • Recombinant Proteins
  • Treatment Outcome
  • Viral Envelope Proteins / genetics
  • Viral Load*
  • Viral Proteins / genetics

Substances

  • Antiviral Agents
  • HVR1 protein, Hepatitis C virus
  • Interferon alpha-2
  • Interferon-alpha
  • RNA, Viral
  • Recombinant Proteins
  • Viral Envelope Proteins
  • Viral Proteins
  • glycoprotein E2, Hepatitis C virus