Calcium mobilization from mitochondria in synaptic transmitter release

J Cell Biol. 2003 Nov 10;163(3):441-3. doi: 10.1083/jcb.200309111.

Abstract

Mitochondria can rapidly accumulate and release Ca2+ upon cell stimulation. A paper by Yang and coworkers in this issue reports an unusual form of synaptic potentiation, dependent on Ca2+ release from mitochondria through the Na+/Ca2+ exchanger and triggered by Na+ entry through voltage-gated channels (Yang et al., 2003).

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Review
  • Comment

MeSH terms

  • Animals
  • Calcium / metabolism*
  • Calcium Signaling / physiology
  • Humans
  • Mitochondria / metabolism*
  • Neurotransmitter Agents / metabolism*
  • Presynaptic Terminals / metabolism*
  • Sodium Channels / metabolism
  • Sodium-Calcium Exchanger / metabolism
  • Synaptic Transmission / physiology*

Substances

  • Neurotransmitter Agents
  • Sodium Channels
  • Sodium-Calcium Exchanger
  • Calcium

Grants and funding