The CCAAT enhancer-binding protein alpha (C/EBPalpha) requires a SWI/SNF complex for proliferation arrest

J Biol Chem. 2004 Feb 20;279(8):7353-8. doi: 10.1074/jbc.M312709200. Epub 2003 Dec 4.

Abstract

The transcription factor CCAAT enhancer-binding protein alpha (C/EBPalpha) is a tumor suppressor in myeloid cells and inhibits proliferation in all cell types examined. C/EBPalpha interacts with the SWI/SNF chromatin-remodeling complex during the regulation of differentiation-specific genes. Here we show that C/EBPalpha fails to suppress proliferation in SWI/SNF defective cell lines after knock-down of SWI/SNF core components or after deletion of the SWI/SNF interaction domain in C/EBPalpha, respectively. Reconstitution of SWI/SNF function restores C/EBPalpha-dependent proliferation arrest. Our results show that the anti-proliferation activity of C/EBPalpha critically depends on components of the SWI/SNF core complex and suggest that the functional interaction between SWI/SNF and C/EBPalpha is a prerequisite for proliferation arrest.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Western
  • CCAAT-Enhancer-Binding Protein-alpha / genetics*
  • CCAAT-Enhancer-Binding Protein-alpha / metabolism
  • CCAAT-Enhancer-Binding Protein-alpha / physiology*
  • Cell Cycle
  • Cell Division
  • Cell Line
  • Chromatin / chemistry*
  • Fibroblasts / metabolism
  • Genes, Reporter
  • Genetic Vectors
  • Mice
  • Mice, Inbred BALB C
  • Models, Genetic
  • NIH 3T3 Cells
  • Plasmids / metabolism
  • Protein Isoforms
  • Protein Structure, Tertiary
  • RNA Interference
  • Rats
  • Retroviridae / genetics
  • Time Factors
  • Transfection

Substances

  • CCAAT-Enhancer-Binding Protein-alpha
  • Chromatin
  • Protein Isoforms