Enteropathogenic Escherichia coli type III effectors EspG and EspG2 disrupt the microtubule network of intestinal epithelial cells

Infect Immun. 2005 Jul;73(7):4385-90. doi: 10.1128/IAI.73.7.4385-4390.2005.

Abstract

Enteropathogenic Escherichia coli infection of intestinal epithelial cells leads to localized depletion of the microtubule cytoskeleton, an effect that is dependent on delivery of type III translocated effector proteins EspG and Orf3 (designated EspG2) to the site of depletion. Microtubule depletion involved disruption rather than displacement of microtubules.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / chemistry
  • Caco-2 Cells
  • Escherichia coli / pathogenicity*
  • Escherichia coli Proteins / physiology*
  • Humans
  • Intestinal Mucosa / microbiology*
  • Microtubules / physiology*
  • Protein Transport

Substances

  • Actins
  • Escherichia coli Proteins
  • EspG protein, E coli