Kainic acid lesion of the striatum increases dopamine release but reduces 3-methoxytyramine level

Brain Res. 1992 Feb 14;572(1-2):247-9. doi: 10.1016/0006-8993(92)90477-q.

Abstract

Kainic acid lesion of the striatum leads to the disruption of the striatonigral regulatory loop. Although microdialysis showed an increase in dopamine release, two days after kainic acid injection, 3-methoxytyramine levels are dramatically decreased. This suggests that the postsynaptic membrane-bound catechol-O-methyltransferase (COMT), but not the glial COMT, is responsible for the formation of measured 3-methoxytyramine.

Publication types

  • Comparative Study

MeSH terms

  • 3,4-Dihydroxyphenylacetic Acid / metabolism
  • Animals
  • Catechol O-Methyltransferase / metabolism
  • Corpus Striatum / physiology*
  • Dialysis
  • Dopamine / analogs & derivatives*
  • Dopamine / metabolism*
  • Homovanillic Acid / metabolism
  • Kainic Acid
  • Male
  • Rats
  • Rats, Inbred Strains

Substances

  • 3,4-Dihydroxyphenylacetic Acid
  • Catechol O-Methyltransferase
  • 3-methoxytyramine
  • Kainic Acid
  • Dopamine
  • Homovanillic Acid