Resistin- and Obesity-associated metabolic diseases

Horm Metab Res. 2007 Oct;39(10):710-6. doi: 10.1055/s-2007-985897.

Abstract

The link between obesity and diabetes is strong as well as complex. Fat cells produce many circulating regulators of insulin sensitivity, including pro-inflammatory cytokines. In rodents, resistin is produced by adipose tissue, and is a significant regulator of glucose metabolism and insulin sensitivity. In humans, resistin is derived made mainly from macrophages. Given the emerging interrelationship between inflammation and metabolic disease, hyperresistinemia may be a biomarker, and/or a mediator, of metabolic and inflammatory diseases in humans as well as in rodents.

Publication types

  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Adipose Tissue / physiology
  • Animals
  • Animals, Genetically Modified
  • Diabetes Mellitus, Type 2 / complications
  • Endocrine System / physiology
  • Humans
  • Inflammation / complications
  • Insulin Resistance / genetics
  • Insulin Resistance / physiology
  • Metabolic Diseases / etiology*
  • Obesity / complications*
  • PPAR gamma / genetics
  • PPAR gamma / physiology
  • Resistin / genetics
  • Resistin / physiology*
  • Rodentia
  • Signal Transduction / genetics
  • Vascular Diseases / etiology
  • Vascular Diseases / genetics

Substances

  • PPAR gamma
  • Resistin