Minocycline-induced DRESS: evidence for accumulation of the culprit drug

Dermatology. 2008;216(3):200-4. doi: 10.1159/000112926. Epub 2008 Jan 9.

Abstract

Background: Minocycline-induced drug rash with eosinophilia and systemic symptoms (DRESS) may have a prolonged course, especially in African and African-American patients.

Objectives: To determine if a prolonged course of minocycline-induced DRESS was associated with an accumulation of the culprit drug.

Patients and methods: We determined plasma and skin levels of minocycline in patients with minocycline-induced DRESS. We investigated the genetic polymorphisms of enzymes potentially involved in the detoxification of the drug, glutathione S-transferases and UDP-glucuronosyltransferases.

Results and conclusions: We demonstrated the persistence of minocycline in the plasma and/or in the skin of 7 out of 9 patients with skin phototypes V-VI. As pigmented skin contains more melanin, this could promote the formation of a melanin-minocycline complex, which could explain the severe and prolonged DRESS which may occur in this subgroup of patients.

MeSH terms

  • Acne Vulgaris / drug therapy
  • Adolescent
  • Adult
  • Anti-Bacterial Agents / adverse effects*
  • Anti-Bacterial Agents / pharmacokinetics
  • Black People*
  • Black or African American
  • Chromatography, High Pressure Liquid
  • Drug Hypersensitivity / enzymology
  • Drug Hypersensitivity / ethnology
  • Drug Hypersensitivity / genetics*
  • Eosinophilia / chemically induced*
  • Eosinophilia / ethnology
  • Eosinophilia / genetics
  • Female
  • Genetic Predisposition to Disease
  • Genotype
  • Glutathione Transferase / genetics*
  • Humans
  • Male
  • Melanins / analysis
  • Middle Aged
  • Minocycline / adverse effects*
  • Minocycline / pharmacokinetics
  • Polymerase Chain Reaction
  • Polymorphism, Genetic
  • Sequence Deletion
  • Skin / pathology
  • Skin Pigmentation
  • Syndrome

Substances

  • Anti-Bacterial Agents
  • Melanins
  • Glutathione Transferase
  • Minocycline