Combining short-term metformin treatment and one bout of exercise does not increase insulin action in insulin-resistant individuals

Am J Physiol Endocrinol Metab. 2010 Apr;298(4):E815-23. doi: 10.1152/ajpendo.00517.2009. Epub 2010 Jan 13.

Abstract

Results from the Diabetes Prevention Program highlight the effectiveness of metformin or regular physical activity in the prevention of type 2 diabetes. Independently, metformin and exercise increase insulin sensitivity, but they have not been studied in combination. To assess the combined effects, insulin-resistant subjects (n = 9) matched for weight, body fat, and aerobic fitness were studied before any treatment (B), after 2-3 wk of 2,000 mg/day metformin (MET), and after metformin plus 40 min of exercise at 65% Vo(2peak) (MET + Ex). A second group (n = 7) was studied at baseline and after an identical bout of exercise with no metformin (Ex). Biopsies of the vastus lateralis were taken at B, after MET, immediately after MET + Ex (group 1), or immediately after Ex (group 2). Insulin sensitivity was assessed 4 h postexercise with a euglycemic hyperinsulinemic (40 mU.m(2).min(-1)) clamp enriched with [6,6-(2)H]glucose. Insulin sensitivity was 54% higher after Ex (P < 0.01), but there was no change with Met + Ex. Skeletal muscle AMPKalpha2 activity was elevated threefold (P < 0.01) after Ex, but there was no increase with MET + Ex. These findings suggest that the combination of short-term metformin treatment and an acute bout of exercise does not enhance insulin sensitivity, and the addition of metformin may attenuate the well-documented effects of exercise alone.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • AMP-Activated Protein Kinases / metabolism
  • Adult
  • Blotting, Western
  • Exercise / physiology*
  • Female
  • Glucose Clamp Technique
  • Heart Rate / physiology
  • Humans
  • Hypoglycemic Agents / pharmacology*
  • Insulin / pharmacology*
  • Insulin / physiology*
  • Insulin Resistance / physiology*
  • Lactic Acid / blood
  • Male
  • Metformin / pharmacology*
  • Middle Aged
  • Muscle, Skeletal / metabolism
  • Oxygen Consumption / drug effects
  • Phosphorylation

Substances

  • Hypoglycemic Agents
  • Insulin
  • Lactic Acid
  • Metformin
  • Prkaa2 protein, mouse
  • AMP-Activated Protein Kinases