Effect of oral atorvastatin on CD4+CD25+ regulatory T cells, FoxP3 expression, and prognosis in patients with ST-segment elevated myocardial infarction before primary percutaneous coronary intervention

J Cardiovasc Pharmacol. 2011 May;57(5):536-41. doi: 10.1097/FJC.0b013e318211d016.

Abstract

Objective: Our objective was to explore the effects of atorvastatin on changes of CD4+CD25+ regulatory T cells (Tregs), FoxP3 expression in the infarct-related coronary artery, and peripheral venous blood of patients with ST-segment elevation myocardial infarction.

Methods: We recorded 112 cases of patients with ST-segment elevation myocardial infarction who were randomly assigned to receive either atorvastatin 80 mg (n = 52) or placebo (n = 60) before primary percutaneous coronary intervention. Blood samples were obtained from the infarct-related coronary artery and peripheral vein during percutaneous coronary intervention. The proportion of CD4+CD25+ Tregs, FoxP3 mRNA expression in blood and concentrations of transforming growth factor-β and interferon-γ in plasma of the samples were measured or detected by flow cytometry, real-time polymerase chain reaction, or enzyme-linked immunosorbent assay, respectively.

Results: In comparison with the control group, the proportions of CD4+CD25+ Tregs and the mRNA level of FoxP3 and transforming growth factor-β significantly increased; however, interferon-γ decreased with atorvastatin therapy. In the controls, the proportions of CD4+CD25+ Tregs and the mRNA level of FoxP3 and transforming growth factor-β were significantly decreased, but the level of interferon-γ increased more in the infarct-related coronary artery than in the peripheral vein.

Conclusion: : The inhibition of CD4+CD25+ Tregs in patients with ST-segment elevation myocardial infarction could be regulated with atorvastatin given before percutaneous coronary intervention.

Publication types

  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Angioplasty, Balloon, Coronary*
  • Atorvastatin
  • CD4 Antigens / immunology
  • Coronary Vessels / immunology
  • Coronary Vessels / metabolism
  • Coronary Vessels / pathology
  • Electrocardiography
  • Female
  • Flow Cytometry
  • Forkhead Transcription Factors / biosynthesis*
  • Heptanoic Acids / administration & dosage
  • Heptanoic Acids / therapeutic use*
  • Humans
  • Immunologic Factors / administration & dosage
  • Immunologic Factors / therapeutic use*
  • Interferon-gamma / blood
  • Interleukin-2 Receptor alpha Subunit / immunology
  • Lymphocyte Count
  • Male
  • Middle Aged
  • Myocardial Infarction / drug therapy*
  • Myocardial Infarction / immunology*
  • Myocardial Infarction / mortality
  • Myocardial Infarction / surgery
  • Prognosis
  • Pyrroles / administration & dosage
  • Pyrroles / therapeutic use*
  • T-Lymphocytes, Regulatory / immunology*
  • Transforming Growth Factor beta / blood
  • Treatment Outcome

Substances

  • CD4 Antigens
  • FOXP3 protein, human
  • Forkhead Transcription Factors
  • Heptanoic Acids
  • IL2RA protein, human
  • Immunologic Factors
  • Interleukin-2 Receptor alpha Subunit
  • Pyrroles
  • Transforming Growth Factor beta
  • Interferon-gamma
  • Atorvastatin