PD-1 expression by canine T cells and functional effects of PD-1 blockade

Vet Comp Oncol. 2017 Dec;15(4):1487-1502. doi: 10.1111/vco.12294. Epub 2017 Jan 25.

Abstract

The co-inhibitory checkpoint molecule programmed death receptor 1 (PD-1) can trigger T cell functional exhaustion upon binding to its ligand PD-L1 expressed on tumour cells or macrophages. PD-1 blocking antibodies have generated remarkable results in human cancer patients, including inducing durable responses in a number of advanced cancers. Therefore, monoclonal antibodies specific for canine PD-1 were assessed for T cell binding and induction of functional activation. A total of 5-10% of CD4 T cells and 20-25% of CD8 T cells from healthy dogs expressed PD-1, and PD-1 expression was upregulated on T cells from dogs with cancer. Functionally, PD-1 antibodies significantly enhanced T-cell activation, as assessed by proliferation and interferon-gamma (IFN-γ) production. PD-1 antibodies also reversed T-cell suppression induced by canine soluble PD-L1 and by tumour cells and tumour explant fragments. These findings indicate that PD-1 antibodies have potential for use in cancer immunotherapy in dogs.

Keywords: T cell; cytokine; immune; tumour.

MeSH terms

  • Animals
  • Blotting, Western / veterinary
  • CD4-Positive T-Lymphocytes / metabolism
  • CD8-Positive T-Lymphocytes / metabolism
  • Dogs
  • Flow Cytometry / veterinary
  • Interferon-gamma / metabolism
  • Myeloid Cells / metabolism
  • Programmed Cell Death 1 Receptor / antagonists & inhibitors
  • Programmed Cell Death 1 Receptor / metabolism*
  • T-Lymphocytes / metabolism*

Substances

  • Programmed Cell Death 1 Receptor
  • Interferon-gamma