Negative 11C-PIB PET Predicts Lack of Alzheimer's Disease Pathology in Postmortem Examination

J Alzheimers Dis. 2018;63(1):79-85. doi: 10.3233/JAD-170569.

Abstract

Our aim was to assess whether in vivo11C-PIB negative memory-impaired subjects may nonetheless exhibit brain Alzheimer's disease (AD) pathology. We re-evaluated the PET images and systematically characterized the postmortem neuropathology of six individuals who had undergone clinically indicated amyloid PET. The single case with negligible amyloid-β (Aβ) pathology had Lewy body disease, where concomitant AD changes are often seen. Further, the subject's plaques were predominantly diffuse. The predictive value of a negative 11C-PIB scan appears to be good, even in memory-impaired populations. Our results suggest that considerable neuritic Aβ plaque pathology in the absence of specific/cortical 11C-PIB binding upon PET is unlikely.

Keywords: Aβ; PIB; amyloid; immunohistochemistry; neuritic plaque; neuropathology; positron emission tomography.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Alzheimer Disease / diagnostic imaging*
  • Alzheimer Disease / pathology*
  • Amyloid beta-Peptides / metabolism
  • Aniline Compounds
  • Autopsy
  • Benzothiazoles / metabolism*
  • Brain / diagnostic imaging
  • Brain / metabolism
  • DNA-Binding Proteins / metabolism
  • Female
  • Humans
  • Male
  • Middle Aged
  • Positron-Emission Tomography*
  • Sequestosome-1 Protein / metabolism
  • Thiazoles
  • alpha-Synuclein / metabolism
  • tau Proteins / metabolism

Substances

  • 2-(4'-(methylamino)phenyl)-6-hydroxybenzothiazole
  • Amyloid beta-Peptides
  • Aniline Compounds
  • Benzothiazoles
  • DNA-Binding Proteins
  • Sequestosome-1 Protein
  • TARDBP protein, human
  • Thiazoles
  • alpha-Synuclein
  • tau Proteins