Abstract
Tumor-initiating cells (TICs) are particularly efficient at evading detection and elimination by the human immune system. Recent data from Yang and collaborators demonstrate that - at least in preclinical hepatocellular carcinoma models - the immunological privilege of CD49f+ TICs can be limited by targeting CD155, resulting in restored sensitivity to immune checkpoint inhibitors.
Keywords:
CCL4; NK cells; PD-1; TIGIT; myeloid-derived suppressor cells; neutrophils.
Copyright © 2024 Elsevier Ltd. All rights reserved.
MeSH terms
-
Animals
-
Carcinoma, Hepatocellular* / drug therapy
-
Carcinoma, Hepatocellular* / immunology
-
Carcinoma, Hepatocellular* / pathology
-
Carcinoma, Hepatocellular* / therapy
-
Humans
-
Immune Checkpoint Inhibitors* / pharmacology
-
Immune Checkpoint Inhibitors* / therapeutic use
-
Immune Evasion*
-
Liver Neoplasms* / drug therapy
-
Liver Neoplasms* / immunology
-
Liver Neoplasms* / pathology
-
Liver Neoplasms* / therapy
-
Neoplastic Stem Cells* / drug effects
-
Neoplastic Stem Cells* / immunology
-
Tumor Escape*
Substances
-
Immune Checkpoint Inhibitors