Thirteen cases support the clinical significance of imprinting center 1 (IC1) microdeletions in Beckwith-Wiedemann syndrome

Clin Epigenetics. 2025 Apr 29;17(1):67. doi: 10.1186/s13148-025-01873-5.

Abstract

Most Beckwith-Wiedemann syndrome (BWS) cases are sporadic; nonetheless, imprinting center 1 (IC1) microdeletions have been suggested as a rare cause of familial BWS, with ~ 20 reported cases. We report 13 cases from nine families with IC1 microdeletions. Recurrent 1.4-kb, 1.8-kb, and 2.2-kb deletions were observed. IC1 hypermethylation was identified in all families, and we established a statistically significant relationship between IC1 microdeletions and hypermethylation (OR: 108.17, p = 2.76e-13). This study confirms IC1 microdeletions as a cause of familial BWS, expands the understanding of their molecular mechanisms, and supports a Likely Pathogenic clinical classification for IC1 microdeletions.

Keywords: BWS; Beckwith–Wiedemann syndrome; Familial BWS; IC1 hypermethylation; IC1 microdeletion; Imprinting center 1; Imprinting disorder; MS-MLPA.

MeSH terms

  • Beckwith-Wiedemann Syndrome* / genetics
  • Child
  • Child, Preschool
  • Clinical Relevance
  • DNA Methylation
  • Female
  • Genomic Imprinting*
  • Humans
  • Infant
  • Male