Genomic landscape of virus-associated cancers

Nat Commun. 2025 Jul 1;16(1):5887. doi: 10.1038/s41467-025-60836-9.

Abstract

It has been estimated that 15%-20% of human cancers are attributable to infections, mostly by carcinogenic viruses. The incidence varies worldwide, with a majority affecting developing countries. Here, we conduct a comparative analysis of virus-positive and virus-negative tumors in nine cancers linked to five viruses. We observe a higher frequency of virus-positive tumors in males, with notable geographic differences in incidence. Our genomic analysis of 1971 tumors reveals a lower somatic burden, distinct mutation signatures, and driver gene mutations in virus-positive tumors. Compared to virus-negative cases, virus-positive cases have fewer mutations of TP53, CDKN2A, and deletions of 9p21.3/CDKN2A-CDKN1A while exhibiting more mutations in RNA helicases DDX3X and EIF4A1. Furthermore, an analysis of clinical trials of PD-(L)1 inhibitors suggests an association of virus-positivity with higher treatment response rate, particularly evident in gastric cancer and head and neck squamous cell carcinoma. Both cancer types also show evidence of increased CD8 + T cell infiltration and T cell receptor clonal selection in virus-positive tumors. These results illustrate the epidemiological, genetic, and therapeutic trends across virus-associated malignancies.

MeSH terms

  • CD8-Positive T-Lymphocytes / immunology
  • Cyclin-Dependent Kinase Inhibitor p16 / genetics
  • Female
  • Genomics
  • Humans
  • Male
  • Mutation
  • Neoplasms* / drug therapy
  • Neoplasms* / epidemiology
  • Neoplasms* / genetics
  • Neoplasms* / virology
  • Oncogenic Viruses* / genetics
  • Tumor Suppressor Protein p53 / genetics

Substances

  • Cyclin-Dependent Kinase Inhibitor p16
  • Tumor Suppressor Protein p53