The tyrosine kinase inhibitor methyl 2,5-dihydroxycinnimate disrupts changes in the actin cytoskeleton required for neurite formation

Brain Res Mol Brain Res. 1996 Dec 31;43(1-2):338-40. doi: 10.1016/s0169-328x(96)00221-5.

Abstract

In the current studies, we investigated the relationship between tyrosine phosphorylation and neurite formation. In SH-SY5Y neuroblastoma cells, the tyrosine kinase inhibitor methyl 2, 5-dihydroxycinnimate blocked neurite formation on laminin. This corresponded with inhibition of paxillin and focal adhesion kinase tyrosine phosphorylation as well as a disruption of actin filament organization and actin polymerization. This suggests that tyrosine phosphorylation helps direct changes in the actin cytoskeleton required for neurite formation.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Actins / metabolism*
  • Cells, Cultured
  • Cinnamates / pharmacology*
  • Cytoskeleton / drug effects*
  • Dose-Response Relationship, Drug
  • Humans
  • Neurites / drug effects*

Substances

  • Actins
  • Cinnamates
  • methyl 2,5-dihydroxycinnamate