Lack of phospholipase A2 mutations in neuroblastoma, melanoma and colon-cancer cell lines

Int J Cancer. 1997 Jul 17;72(2):337-9. doi: 10.1002/(sici)1097-0215(19970717)72:2<337::aid-ijc22>3.0.co;2-b.

Abstract

A candidate murine tumor-suppressor gene, Mom1, has been identified as the secretory phospholipase A2 (GDB nomenclature: PLA2G2A) gene. Evidence suggests that PLA2G2A functions as a tumor-suppressor because mice lacking PLA2G2A expression demonstrate increased colonic polyposis. The human homologue of PLA2G2A has been mapped to chromosome 1p36, a region frequently implicated in the pathogenesis of neuroblastoma, colon cancer and melanoma. We identified 2 alterations in the PLA2G2A gene in a single neuroblastoma cell line out of 20 examined; however, we found no mutations in 24 melanoma cell lines, 12 lymphoblastoid cell lines from patients having chromosome 1-linked familial melanoma and 10 colon cancer cell lines. Secretory phospholipase A2 is unlikely to play a significant role in the pathogenesis of these tumors.

MeSH terms

  • Animals
  • Brain Neoplasms / genetics*
  • Chromosome Mapping
  • Chromosomes, Human, Pair 1*
  • Colonic Neoplasms / genetics*
  • Genes, Tumor Suppressor
  • Humans
  • Melanoma / genetics*
  • Mice
  • Mutation*
  • Neuroblastoma / genetics*
  • Phospholipases A / genetics*
  • Phospholipases A2
  • Tumor Cells, Cultured

Substances

  • Phospholipases A
  • Phospholipases A2