Velez-Cruz R - Search Results

1

Deletion of 5' sequences of the CSB gene provides insight into the pathophysiology of Cockayne syndrome.

Laugel V, Dalloz C, Stary A, Cormier-Daire V, Desguerre I, Renouil M, Fourmaintraux A, Velez-Cruz R, Egly JM, Sarasin A, Dollfus H. Laugel V, et al. Eur J Hum Genet. 2008 Mar;16(3):320-7. doi: 10.1038/sj.ejhg.5201991. Epub 2008 Jan 9. Eur J Hum Genet. 2008. PMID: 18183039

2

Cockayne syndrome group B (CSB) protein: at the crossroads of transcriptional networks.

Vélez-Cruz R, Egly JM. Vélez-Cruz R, et al. Mech Ageing Dev. 2013 May-Jun;134(5-6):234-42. doi: 10.1016/j.mad.2013.03.004. Epub 2013 Apr 3. Mech Ageing Dev. 2013. PMID: 23562425 Review.

3

Sirt1 suppresses RNA synthesis after UV irradiation in combined xeroderma pigmentosum group D/Cockayne syndrome (XP-D/CS) cells.

Vélez-Cruz R, Zadorin AS, Coin F, Egly JM. Vélez-Cruz R, et al. Proc Natl Acad Sci U S A. 2013 Jan 15;110(3):E212-20. doi: 10.1073/pnas.1213076110. Epub 2012 Dec 24. Proc Natl Acad Sci U S A. 2013. PMID: 23267107 Free PMC article.

4

CSA and CSB proteins interact with p53 and regulate its Mdm2-dependent ubiquitination.

Latini P, Frontini M, Caputo M, Gregan J, Cipak L, Filippi S, Kumar V, Vélez-Cruz R, Stefanini M, Proietti-De-Santis L. Latini P, et al. Cell Cycle. 2011 Nov 1;10(21):3719-30. doi: 10.4161/cc.10.21.17905. Epub 2011 Nov 1. Cell Cycle. 2011. PMID: 22032989 Free PMC article.

5

The CSB repair factor is overexpressed in cancer cells, increases apoptotic resistance, and promotes tumor growth.

Caputo M, Frontini M, Velez-Cruz R, Nicolai S, Prantera G, Proietti-De-Santis L. Caputo M, et al. Among authors: velez cruz r. DNA Repair (Amst). 2013 Apr 1;12(4):293-9. doi: 10.1016/j.dnarep.2013.01.008. Epub 2013 Feb 16. DNA Repair (Amst). 2013. PMID: 23419237 Free PMC article.

6

NER factors are recruited to active promoters and facilitate chromatin modification for transcription in the absence of exogenous genotoxic attack.

Le May N, Mota-Fernandes D, Vélez-Cruz R, Iltis I, Biard D, Egly JM. Le May N, et al. Mol Cell. 2010 Apr 9;38(1):54-66. doi: 10.1016/j.molcel.2010.03.004. Mol Cell. 2010. PMID: 20385089 Free article.

7

E2F1 and p53 transcription factors as accessory factors for nucleotide excision repair.

Vélez-Cruz R, Johnson DG. Vélez-Cruz R, et al. Int J Mol Sci. 2012 Oct 19;13(10):13554-68. doi: 10.3390/ijms131013554. Int J Mol Sci. 2012. PMID: 23202967 Free PMC article. Review.

8

The SWI/SNF ATPase BRG1 stimulates DNA end resection and homologous recombination by reducing nucleosome density at DNA double strand breaks and by promoting the recruitment of the CtIP nuclease.

Hays E, Nettleton E, Carter C, Morales M, Vo L, Passo M, Vélez-Cruz R. Hays E, et al. Among authors: velez cruz r. Cell Cycle. 2020 Nov;19(22):3096-3114. doi: 10.1080/15384101.2020.1831256. Epub 2020 Oct 12. Cell Cycle. 2020. PMID: 33044911 Free PMC article.

9

The E2F1 transcription factor and RB tumor suppressor moonlight as DNA repair factors.

Manickavinayaham S, Velez-Cruz R, Biswas AK, Chen J, Guo R, Johnson DG. Manickavinayaham S, et al. Among authors: velez cruz r. Cell Cycle. 2020 Sep;19(18):2260-2269. doi: 10.1080/15384101.2020.1801190. Epub 2020 Aug 13. Cell Cycle. 2020. PMID: 32787501 Free PMC article. Review.

10

RB localizes to DNA double-strand breaks and promotes DNA end resection and homologous recombination through the recruitment of BRG1.

Vélez-Cruz R, Manickavinayaham S, Biswas AK, Clary RW, Premkumar T, Cole F, Johnson DG. Vélez-Cruz R, et al. Genes Dev. 2016 Nov 15;30(22):2500-2512. doi: 10.1101/gad.288282.116. Genes Dev. 2016. PMID: 27940962 Free PMC article.

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