Cullin C - Search Results

1

In vivo and in vitro analyses of toxic mutants of HET-s: FTIR antiparallel signature correlates with amyloid toxicity.

Berthelot K, Ta HP, Géan J, Lecomte S, Cullin C. Berthelot K, et al. Among authors: cullin c. J Mol Biol. 2011 Sep 9;412(1):137-52. doi: 10.1016/j.jmb.2011.07.009. Epub 2011 Jul 18. J Mol Biol. 2011. PMID: 21782829

2

Conservation of the prion properties of Ure2p through evolution.

Baudin-Baillieu A, Fernandez-Bellot E, Reine F, Coissac E, Cullin C. Baudin-Baillieu A, et al. Among authors: cullin c. Mol Biol Cell. 2003 Aug;14(8):3449-58. doi: 10.1091/mbc.e03-01-0007. Epub 2003 May 18. Mol Biol Cell. 2003. PMID: 12925776 Free PMC article.

3

The [URE3] yeast prion results from protein aggregates that differ from amyloid filaments formed in vitro.

Ripaud L, Maillet L, Immel-Torterotot F, Durand F, Cullin C. Ripaud L, et al. Among authors: cullin c. J Biol Chem. 2004 Dec 3;279(49):50962-8. doi: 10.1074/jbc.M408792200. Epub 2004 Sep 28. J Biol Chem. 2004. PMID: 15456789 Free article.

4

A novel in vitro filter trap assay identifies tannic acid as an amyloid aggregation inducer for HET-s.

Boyé-Harnasch M, Cullin C. Boyé-Harnasch M, et al. Among authors: cullin c. J Biotechnol. 2006 Sep 1;125(2):222-30. doi: 10.1016/j.jbiotec.2006.03.006. Epub 2006 Apr 18. J Biotechnol. 2006. PMID: 16621084

5

The cellular concentration of the yeast Ure2p prion protein affects its propagation as a prion.

Crapeau M, Marchal C, Cullin C, Maillet L. Crapeau M, et al. Among authors: cullin c. Mol Biol Cell. 2009 Apr;20(8):2286-96. doi: 10.1091/mbc.e08-11-1097. Epub 2009 Feb 18. Mol Biol Cell. 2009. PMID: 19225154 Free PMC article.

6

Driving amyloid toxicity in a yeast model by structural changes: a molecular approach.

Berthelot K, Immel F, Géan J, Lecomte S, Oda R, Kauffmann B, Cullin C. Berthelot K, et al. Among authors: cullin c. FASEB J. 2009 Jul;23(7):2254-63. doi: 10.1096/fj.08-125724. Epub 2009 Feb 19. FASEB J. 2009. PMID: 19228879

7

Screening for toxic amyloid in yeast exemplifies the role of alternative pathway responsible for cytotoxicity.

Couthouis J, Rébora K, Immel F, Berthelot K, Castroviejo M, Cullin C. Couthouis J, et al. Among authors: cullin c. PLoS One. 2009;4(3):e4539. doi: 10.1371/journal.pone.0004539. Epub 2009 Mar 5. PLoS One. 2009. PMID: 19262694 Free PMC article.

8

A yeast toxic mutant of HET-s((218-289)) prion displays alternative intermediates of amyloidogenesis.

Berthelot K, Lecomte S, Géan J, Immel F, Cullin C. Berthelot K, et al. Among authors: cullin c. Biophys J. 2010 Aug 9;99(4):1239-46. doi: 10.1016/j.bpj.2010.06.015. Biophys J. 2010. PMID: 20713008 Free PMC article.

9

The toxicity of an "artificial" amyloid is related to how it interacts with membranes.

Couthouis J, Marchal C, D'Angelo F, Berthelot K, Cullin C. Couthouis J, et al. Among authors: cullin c. Prion. 2010 Oct-Dec;4(4):283-91. doi: 10.4161/pri.4.4.13126. Epub 2010 Oct 23. Prion. 2010. PMID: 21057225 Free PMC article.

10

Comparative studies of nontoxic and toxic amyloids interacting with membrane models at the air-water interface.

Ta HP, Berthelot K, Coulary-Salin B, Desbat B, Géan J, Servant L, Cullin C, Lecomte S. Ta HP, et al. Among authors: cullin c. Langmuir. 2011 Apr 19;27(8):4797-807. doi: 10.1021/la103788r. Epub 2011 Mar 15. Langmuir. 2011. PMID: 21405042

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