Lin YW - Search Results

1

Heterogeneity and function of K(ATP) channels in canine hearts.

Zhang HX, Silva JR, Lin YW, Verbsky JW, Lee US, Kanter EM, Yamada KA, Schuessler RB, Nichols CG. Zhang HX, et al. Among authors: lin yw. Heart Rhythm. 2013 Oct;10(10):1576-83. doi: 10.1016/j.hrthm.2013.07.020. Epub 2013 Jul 17. Heart Rhythm. 2013. PMID: 23871704 Free PMC article.

2

Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits.

Lin YW, Akrouh A, Hsu Y, Hughes N, Nichols CG, De León DD. Lin YW, et al. Channels (Austin). 2012 Mar-Apr;6(2):133-8. doi: 10.4161/chan.19980. Epub 2012 Mar 1. Channels (Austin). 2012. PMID: 22562119 Free PMC article.

3

Functional characterization of a novel KCNJ11 in frame mutation-deletion associated with infancy-onset diabetes and a mild form of intermediate DEND: a battle between K(ATP) gain of channel activity and loss of channel expression.

Lin YW, Li A, Grasso V, Battaglia D, Crinò A, Colombo C, Barbetti F, Nichols CG. Lin YW, et al. PLoS One. 2013 May 7;8(5):e63758. doi: 10.1371/journal.pone.0063758. Print 2013. PLoS One. 2013. PMID: 23667671 Free PMC article.

4

Tuning the electrical properties of the heart by differential trafficking of KATP ion channel complexes.

Arakel EC, Brandenburg S, Uchida K, Zhang H, Lin YW, Kohl T, Schrul B, Sulkin MS, Efimov IR, Nichols CG, Lehnart SE, Schwappach B. Arakel EC, et al. Among authors: lin yw. J Cell Sci. 2014 May 1;127(Pt 9):2106-19. doi: 10.1242/jcs.141440. Epub 2014 Feb 25. J Cell Sci. 2014. PMID: 24569881 Free PMC article.

5

Glyburide ameliorates motor coordination and glucose homeostasis in a child with diabetes associated with the KCNJ11/S225T, del226-232 mutation.

Battaglia D, Lin YW, Brogna C, Crinò A, Grasso V, Mozzi AF, Russo L, Spera S, Colombo C, Ricci S, Nichols CG, Mercuri E, Barbetti F. Battaglia D, et al. Among authors: lin yw. Pediatr Diabetes. 2012 Dec;13(8):656-60. doi: 10.1111/j.1399-5448.2012.00874.x. Epub 2012 Jun 14. Pediatr Diabetes. 2012. PMID: 22694282 Free PMC article.

6

Kir6.2 mutations associated with neonatal diabetes reduce expression of ATP-sensitive K+ channels: implications in disease mechanism and sulfonylurea therapy.

Lin CW, Lin YW, Yan FF, Casey J, Kochhar M, Pratt EB, Shyng SL. Lin CW, et al. Among authors: lin yw. Diabetes. 2006 Jun;55(6):1738-46. doi: 10.2337/db05-1571. Diabetes. 2006. PMID: 16731837

7

Clinical characteristics and biochemical mechanisms of congenital hyperinsulinism associated with dominant KATP channel mutations.

Pinney SE, MacMullen C, Becker S, Lin YW, Hanna C, Thornton P, Ganguly A, Shyng SL, Stanley CA. Pinney SE, et al. Among authors: lin yw. J Clin Invest. 2008 Aug;118(8):2877-86. doi: 10.1172/JCI35414. J Clin Invest. 2008. PMID: 18596924 Free PMC article.

8

Congenital hyperinsulinism associated ABCC8 mutations that cause defective trafficking of ATP-sensitive K+ channels: identification and rescue.

Yan FF, Lin YW, MacMullen C, Ganguly A, Stanley CA, Shyng SL. Yan FF, et al. Among authors: lin yw. Diabetes. 2007 Sep;56(9):2339-48. doi: 10.2337/db07-0150. Epub 2007 Jun 15. Diabetes. 2007. PMID: 17575084 Free PMC article.

9

A novel KCNJ11 mutation associated with congenital hyperinsulinism reduces the intrinsic open probability of beta-cell ATP-sensitive potassium channels.

Lin YW, MacMullen C, Ganguly A, Stanley CA, Shyng SL. Lin YW, et al. J Biol Chem. 2006 Feb 3;281(5):3006-12. doi: 10.1074/jbc.M511875200. Epub 2005 Dec 6. J Biol Chem. 2006. PMID: 16332676 Free PMC article.

10

Stabilization of the activity of ATP-sensitive potassium channels by ion pairs formed between adjacent Kir6.2 subunits.

Lin YW, Jia T, Weinsoft AM, Shyng SL. Lin YW, et al. J Gen Physiol. 2003 Aug;122(2):225-37. doi: 10.1085/jgp.200308822. J Gen Physiol. 2003. PMID: 12885877 Free PMC article.

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